CherryActive Concentrate Antioxidant Testing Results
CherryActive Concentrate has recently been tested for its antioxidant capacity against other products that are considered or marketed as “superfruits” or “high in antioxidants”. This was performed at a leading European antioxidant testing laboratory, Knight Scientific www.knightscientific.com
Comparison of Antioxidant Capacity of CherryActive Concentrate (30mL) Against Single Servings of Other Drinks.
It can be seen that a single 30ml dose of the CherryActive Concentrate provides an antioxidant capacity about 8‐fold greater than the 250ml Innocent Blackberries/Blueberries Smoothie, a product held in high regard for its health-promoting qualities. The antioxidant capacity for the CherryActive Concentrate is one of the highest tested at Knight Scientific for a food supplement product.
Dr Robert Verker BSc MSc DIC PhD, ANH Consultancy Ltd, a leading UK scientific specialist said:
“To put CherryActive’s antioxidant results into perspective, this means that a single 30mL serving gives you about the same free radical quenching ability as over 20 typical portions of fruit and vegetable. The results also show the antioxidant effects work well across 5 different free radical systems, something that can’t be said about simpler antioxidants like single vitamins”.
The antioxidant testing was performed on one of the most destructive of all free radicals, peroxynitrite. A summary of the role of peroxynitrite as a free radical, is given in a recent paper by Pachel et al (Physiol Rev. 2007 January; 87(1): 315–424). The abstract of the paper is given below:
The discovery that mammalian cells have the ability to synthesize the free radical nitric oxide (NO) has stimulated an extraordinary impetus for scientific research in all the fields of biology and medicine. Since its early description as an endothelial‐derived relaxing factor, NO has emerged as a fundamental signaling device regulating virtually every critical cellular function, as well as a potent mediator of cellular damage in a wide range of conditions. Recent evidence indicates that most of the cytotoxicity attributed to NO is rather due to peroxynitrite, produced from the diffusion‐controlled reaction between NO and another free radical, the superoxide anion. Peroxynitrite interacts with lipids, DNA, and proteins via direct oxidative reactions or via indirect, radical‐mediated mechanisms. These reactions trigger cellular responses ranging from subtle modulations of cell signaling to overwhelming oxidative injury, committing cells to necrosis or apoptosis. In vivo, peroxynitrite generation represents a crucial pathogenic mechanism in conditions such as stroke, myocardial infarction, chronic heart failure, diabetes, circulatory shock, chronic inflammatory diseases, cancer, and neurodegenerative disorders. Hence, novel pharmacological strategies aimed at removing peroxynitrite might represent powerful therapeutic tools in the future.